Billets de Michelle Dawson

Billet publié sur Bluesky le 29/05/2026 12:44

Bluesky Publication avec DOI crossref Extrait cité dans le billet Question posée par Dawson dans le billet Lien intégré au billet Termes sur l’autisme

Autistic "hypoconnectivity and hyperconnectivity subtypes"? "a new empirical framework for targeted subtyping of the autism spectrum"?--but only 25% of autistics could be subtyped? www.nature.com/articles/s41... & subtypes did not differ in RRBs, or "age, IQ, sex, psychiatric co-occurrence..."?

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Publication avec DOI Récupéré Publication crossref

Marco Pagani, Valerio Zerbi, Silvia Gini, Filomena Grazia Alvino, Abhishek Banerjee, Andrea Barberis, M. Albert Basson, Yuri Bozzi, et al. (2026). Autism subtypes identified using cross-species functional connectivity analyses. Nature Neuroscience, 29(6), 1476-1487. Springer Science and Business Media LLC.

Date de publication
15/05/2026
Identifiant
10.1038/s41593-026-02287-z
Auteurs
Marco Pagani, Valerio Zerbi, Silvia Gini, Filomena Grazia Alvino, Abhishek Banerjee, Andrea Barberis, M. Albert Basson, Yuri Bozzi, Alberto Galbusera, Jacob Ellegood, Michela Fagiolini, Jason P. Lerch, Michela Matteoli, Caterina Montani, Davide Pozzi, Giovanni Provenzano, Maria Luisa Scattoni, Nicole Wenderoth, Ting Xu, Michael V. Lombardo, Michael P. Milham, Adriana Di Martino, Alessandro Gozzi
Source
Nature Neuroscience
Détails
29(6), 1476-1487
Type de référence
article
Éditeur
Springer Science and Business Media LLC
Source de métadonnées
crossref

Résumé

Abstract It is often assumed that phenotypic heterogeneity in autism reflects underlying pathobiological variation. However, direct evidence supporting this link is lacking. Leveraging cross-species functional neuroimaging, we show that brain dysconnectivity patterns in autism can be parsed into biologically dissociable subtypes. Specifically, we found that functional magnetic resonance imaging (fMRI) connectivity alterations in 20 distinct genetic mouse models of autism cluster into hypoconnectivity-dominant and hyperconnectivity-dominant subtypes. These subtypes are linked to distinct biological pathways, with hypoconnectivity being associated with synaptic dysfunction and hyperconnectivity reflecting transcriptional and immune-related alterations. Here we identified analogous hypoconnectivity and hyperconnectivity subtypes in a multicenter human fMRI dataset of n = 940 individuals with idiopathic autism and n = 1,036 neurotypical individuals. The human autism subtypes are highly replicable, are associated with distinct functional network architectures and behavioral profiles and recapitulate the synaptic and immune-related pathways identified in the rodent dataset. Our work provides a new empirical framework for targeted subtyping of the autism spectrum.

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